Sabet, Nima Shokouhi <br/><br/>
Curcumin attenuates bevacizumab-induced toxicity via suppressing oxidative stress and preventing mitochondrial dysfunction in heart mitochondria.  [electronic resource] 

 -  Naunyn-Schmiedeberg's archives of pharmacology  08 2020 
 - 1447-1457 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1432-1912 
<br/><br/><label>Standard No.: </label>10.1007/s00210-020-01853-x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Angiogenesis Inhibitors--toxicity<br/>Animals<br/>Antioxidants--pharmacology<br/>Apoptosis--drug effects<br/>Bevacizumab--toxicity<br/>Cardiotoxicity<br/>Curcumin--pharmacology<br/>Heart Failure--chemically induced<br/>Lipid Peroxidation--drug effects<br/>Male<br/>Membrane Potential, Mitochondrial--drug effects<br/>Mitochondria, Heart--drug effects<br/>Mitochondrial Swelling--drug effects<br/>Myocytes, Cardiac--drug effects<br/>Oxidative Stress--drug effects<br/>Rats, Wistar<br/>Reactive Oxygen Species--metabolism