Cheng, Xiaoli <br/><br/>
18α-Glycyrrhetinic acid (GA) ameliorates fructose-induced nephropathy in mice by suppressing oxidative stress, dyslipidemia and inflammation.  [electronic resource] 

 -  Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie  May 2020 
 - 109702 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1950-6007 
<br/><br/><label>Standard No.: </label>10.1016/j.biopha.2019.109702  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acute Kidney Injury--chemically induced<br/>Animals<br/>Anti-Inflammatory Agents--pharmacology<br/>Cell Survival--drug effects<br/>Cytokines--metabolism<br/>Dyslipidemias--metabolism<br/>Fatty Acids--metabolism<br/>Fructose--pharmacology<br/>Glycyrrhetinic Acid--analogs & derivatives<br/>Glycyrrhiza--chemistry<br/>Humans<br/>Inflammation--metabolism<br/>MAP Kinase Signaling System--drug effects<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mitochondria--pathology<br/>NF-E2-Related Factor 2--metabolism<br/>NF-kappa B--metabolism<br/>Oxidative Stress--drug effects