Tanaka, Hikari <br/><br/>
YAP-dependent necrosis occurs in early stages of Alzheimer's disease and regulates mouse model pathology.  [electronic resource] 

 -  Nature communications  01 2020 
 - 507 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2041-1723 
<br/><br/><label>Standard No.: </label>10.1038/s41467-020-14353-6  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adaptor Proteins, Signal Transducing--metabolism<br/>Alzheimer Disease--cerebrospinal fluid<br/>Amyloid beta-Peptides--metabolism<br/>Animals<br/>Cell Cycle Proteins--metabolism<br/>Cell Nucleus--metabolism<br/>Cognitive Dysfunction--cerebrospinal fluid<br/>Computer Simulation<br/>Disease Models, Animal<br/>Endoplasmic Reticulum--pathology<br/>Female<br/>HMGB1 Protein--cerebrospinal fluid<br/>Humans<br/>Induced Pluripotent Stem Cells--metabolism<br/>Lysophospholipids--metabolism<br/>Male<br/>Mice, Transgenic<br/>Necrosis<br/>Neurons--metabolism<br/>Signal Transduction<br/>Sphingosine--analogs & derivatives<br/>Time-Lapse Imaging<br/>Transcription Factors--metabolism<br/>YAP-Signaling Proteins