Sparkenbaugh, Erica M <br/><br/>
Thrombin activation of PAR-1 contributes to microvascular stasis in mouse models of sickle cell disease.  [electronic resource] 

 -  Blood  05 2020 
 - 1783-1787 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1528-0020 
<br/><br/><label>Standard No.: </label>10.1182/blood.2019003543  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Anemia, Sickle Cell--complications<br/>Animals<br/>Blood Coagulation Disorders--etiology<br/>Blood Platelets--metabolism<br/>Constriction, Pathologic--genetics<br/>Disease Models, Animal<br/>Female<br/>Hemoglobin, Sickle--genetics<br/>Humans<br/>Male<br/>Mice<br/>Mice, Transgenic<br/>Microvessels--metabolism<br/>Receptor, PAR-1--genetics<br/>Thrombin--metabolism<br/>Vascular Diseases--etiology