Yu, Jiao-Jiao <br/><br/>
Disruption of the EGFR-SQSTM1 interaction by a stapled peptide suppresses lung cancer via activating autophagy and inhibiting EGFR signaling.  [electronic resource] 

 -  Cancer letters  04 2020 
 - 23-35 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1872-7980 
<br/><br/><label>Standard No.: </label>10.1016/j.canlet.2020.01.004  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antineoplastic Agents--pharmacology<br/>Apoptosis<br/>Autophagy<br/>Biomarkers, Tumor--genetics<br/>Carcinoma, Non-Small-Cell Lung--drug therapy<br/>Cell Proliferation<br/>Drug Resistance, Neoplasm<br/>ErbB Receptors--antagonists & inhibitors<br/>Gene Expression Regulation, Neoplastic<br/>Humans<br/>Lung Neoplasms--drug therapy<br/>Male<br/>Mice<br/>Mice, Inbred BALB C<br/>Mice, Nude<br/>Mutation<br/>Peptide Fragments--pharmacology<br/>Protein Kinase Inhibitors--pharmacology<br/>Protein Multimerization<br/>Sequestosome-1 Protein--antagonists & inhibitors<br/>Signal Transduction<br/>Tumor Cells, Cultured<br/>Xenograft Model Antitumor Assays