Hu, Dong <br/><br/>
Human ucMSCs seeded in a decellularized kidney scaffold attenuate renal fibrosis by reducing epithelial-mesenchymal transition via the TGF-β/Smad signaling pathway.  [electronic resource] 

 -  Pediatric research  08 2020 
 - 192-201 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1530-0447 
<br/><br/><label>Standard No.: </label>10.1038/s41390-019-0736-6  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cells, Cultured<br/>Cytokines--metabolism<br/>Epithelial-Mesenchymal Transition<br/>Extracellular Matrix--metabolism<br/>Fibrosis--physiopathology<br/>Gene Expression Profiling<br/>Humans<br/>Inflammation<br/>Kidney--physiopathology<br/>Kidney Transplantation<br/>Male<br/>Mesenchymal Stem Cell Transplantation--methods<br/>Mesenchymal Stem Cells--cytology<br/>Microscopy, Electron, Scanning<br/>Nephrectomy<br/>Porosity<br/>Rats<br/>Rats, Sprague-Dawley<br/>Renal Insufficiency<br/>Signal Transduction<br/>Smad Proteins--biosynthesis<br/>Tissue Scaffolds<br/>Transforming Growth Factor beta1--biosynthesis<br/>Umbilical Cord--cytology<br/>Urinary Tract--metabolism