Yang, Dongqiang <br/><br/>
TLR4 induced Wnt3a-Dvl3 restrains the intensity of inflammation and protects against endotoxin-driven organ failure through GSK3β/β-catenin signaling.  [electronic resource] 

 -  Molecular immunology  02 2020 
 - 153-164 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1872-9142 
<br/><br/><label>Standard No.: </label>10.1016/j.molimm.2019.12.013  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cytokines--metabolism<br/>Dishevelled Proteins--metabolism<br/>Endotoxins--metabolism<br/>Glycogen Synthase Kinase 3 beta--metabolism<br/>Humans<br/>Inflammation--metabolism<br/>Lipopolysaccharides--pharmacology<br/>Mice<br/>Mice, Inbred C57BL<br/>Monocytes--metabolism<br/>Multiple Organ Failure--metabolism<br/>Phosphorylation--physiology<br/>Signal Transduction--physiology<br/>Toll-Like Receptor 4--metabolism<br/>Transcription Factor RelA--metabolism<br/>Tumor Necrosis Factor-alpha--metabolism<br/>Wnt3A Protein--metabolism<br/>beta Catenin--metabolism