Stevens, Alexandra M <br/><br/>
Atovaquone is active against AML by upregulating the integrated stress pathway and suppressing oxidative phosphorylation.  [electronic resource] 

 -  Blood advances  12 2019 
 - 4215-4227 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>2473-9537 
<br/><br/><label>Standard No.: </label>10.1182/bloodadvances.2019000499  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Activating Transcription Factor 4--metabolism<br/>Adolescent<br/>Animals<br/>Apoptosis--drug effects<br/>Atovaquone--pharmacology<br/>Cell Line, Tumor<br/>Cell Survival--drug effects<br/>Child<br/>Child, Preschool<br/>Disease Models, Animal<br/>Female<br/>Humans<br/>Infant<br/>Leukemia, Myeloid, Acute--genetics<br/>Male<br/>Mice<br/>Mice, Knockout<br/>Oxidative Phosphorylation--drug effects<br/>Signal Transduction--drug effects<br/>Xenograft Model Antitumor Assays