Amyloid Beta and Tau Cooperate to Cause Reversible Behavioral and Transcriptional Deficits in a Model of Alzheimer's Disease. [electronic resource]
- Cell reports 12 2019
- 3592-3604.e5 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
2211-1247
10.1016/j.celrep.2019.11.044 doi
Aged Aged, 80 and over Alzheimer Disease--metabolism Amyloid beta-Peptides--metabolism Animals Female Humans Male Mice Microglia--metabolism Spatial Behavior Synapses--metabolism Transcriptome tau Proteins--metabolism