Benzoni, Patrizia <br/><br/>
Human iPSC modelling of a familial form of atrial fibrillation reveals a gain of function of If and ICaL in patient-derived cardiomyocytes.  [electronic resource] 

 -  Cardiovascular research  05 2020 
 - 1147-1160 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1755-3245 
<br/><br/><label>Standard No.: </label>10.1093/cvr/cvz217  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Action Potentials--drug effects<br/>Anti-Arrhythmia Agents--therapeutic use<br/>Atrial Fibrillation--drug therapy<br/>Calcium Channels, L-Type--genetics<br/>Case-Control Studies<br/>Cell Differentiation<br/>Cells, Cultured<br/>Drug Resistance--genetics<br/>Genetic Predisposition to Disease<br/>Heart Rate--drug effects<br/>Humans<br/>Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels--genetics<br/>Induced Pluripotent Stem Cells--metabolism<br/>Middle Aged<br/>Mutation<br/>Myocytes, Cardiac--metabolism<br/>Siblings<br/>Exome Sequencing