Nakagawa, Takatoshi <br/><br/>
Augmented O-GlcNAcylation attenuates intermittent hypoxia-induced cardiac remodeling through the suppression of NFAT and NF-κB activities in mice.  [electronic resource] 

 -  Hypertension research : official journal of the Japanese Society of Hypertension  12 2019 
 - 1858-1871 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1348-4214 
<br/><br/><label>Standard No.: </label>10.1038/s41440-019-0311-x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acylation<br/>Animals<br/>Cell Line<br/>Diabetes Mellitus, Type 2--complications<br/>Echocardiography<br/>Glycogen Synthase Kinase 3 beta--metabolism<br/>HEK293 Cells<br/>Humans<br/>Hypoxia--pathology<br/>Mice<br/>Mice, Transgenic<br/>Myocardium--metabolism<br/>N-Acetylglucosaminyltransferases--genetics<br/>NF-kappa B--metabolism<br/>NFATC Transcription Factors--metabolism<br/>Oxidative Stress<br/>Phosphorylation<br/>Ventricular Remodeling