Wu, Han-Fang <br/><br/>
Targeting the inhibition of fatty acid amide hydrolase ameliorate the endocannabinoid-mediated synaptic dysfunction in a valproic acid-induced rat model of Autism.  [electronic resource] 

 -  Neuropharmacology  01 2020 
 - 107736 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1873-7064 
<br/><br/><label>Standard No.: </label>10.1016/j.neuropharm.2019.107736  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amidohydrolases--antagonists & inhibitors<br/>Animals<br/>Arachidonic Acids--metabolism<br/>Autism Spectrum Disorder--chemically induced<br/>Benzamides--pharmacology<br/>Carbamates--pharmacology<br/>Disease Models, Animal<br/>Endocannabinoids--metabolism<br/>Enzyme Inhibitors--toxicity<br/>Female<br/>Long-Term Synaptic Depression--drug effects<br/>Neuronal Plasticity--drug effects<br/>Polyunsaturated Alkamides--metabolism<br/>Prefrontal Cortex--metabolism<br/>Pregnancy<br/>Prenatal Exposure Delayed Effects--chemically induced<br/>Protein Transport--drug effects<br/>Rats<br/>Receptors, AMPA--drug effects<br/>Receptors, Metabotropic Glutamate--drug effects<br/>Social Behavior<br/>Valproic Acid--toxicity