Huang, Juan <br/><br/>
Ghrelin attenuates oxidative stress and neuronal apoptosis via GHSR-1α/AMPK/Sirt1/PGC-1α/UCP2 pathway in a rat model of neonatal HIE.  [electronic resource] 

 -  Free radical biology & medicine  09 2019 
 - 322-337 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1873-4596 
<br/><br/><label>Standard No.: </label>10.1016/j.freeradbiomed.2019.07.001  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>AMP-Activated Protein Kinase Kinases<br/>Animals<br/>Animals, Newborn<br/>Apoptosis--drug effects<br/>Disease Models, Animal<br/>Ghrelin--pharmacology<br/>Humans<br/>Hypoxia-Ischemia, Brain--drug therapy<br/>Neurons--drug effects<br/>Oxidative Stress--drug effects<br/>Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha--genetics<br/>Protein Kinases--genetics<br/>Rats<br/>Receptors, Ghrelin--genetics<br/>Signal Transduction--drug effects<br/>Sirtuin 1--genetics<br/>Uncoupling Protein 2--genetics