Cai, Hua-An <br/><br/>
Ginsenoside (Rg-1) promoted the wound closure of diabetic foot ulcer through iNOS elevation via miR-23a/IRF-1 axis.  [electronic resource] 

 -  Life sciences  Sep 2019 
 - 116525 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1879-0631 
<br/><br/><label>Standard No.: </label>10.1016/j.lfs.2019.05.081  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis<br/>Cell Movement<br/>Cell Proliferation<br/>Central Nervous System Agents--pharmacology<br/>Diabetes Mellitus, Experimental--complications<br/>Diabetic Foot--drug therapy<br/>Gene Expression Regulation<br/>Ginsenosides--pharmacology<br/>Human Umbilical Vein Endothelial Cells<br/>Humans<br/>Interferon Regulatory Factor-1--genetics<br/>Male<br/>MicroRNAs--genetics<br/>Nitric Oxide Synthase Type II--genetics<br/>Rats<br/>Rats, Sprague-Dawley<br/>Wound Healing--drug effects