Wang, Liping <br/><br/>
α2,6-Sialylation promotes immune escape in hepatocarcinoma cells by regulating T cell functions and CD147/MMP signaling.  [electronic resource] 

 -  Journal of physiology and biochemistry  Jun 2019 
 - 199-207 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1877-8755 
<br/><br/><label>Standard No.: </label>10.1007/s13105-019-00674-8  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antigens, CD--metabolism<br/>Apoptosis<br/>Basigin--metabolism<br/>CD8-Positive T-Lymphocytes--immunology<br/>Carcinoma, Hepatocellular--enzymology<br/>Cell Line, Tumor<br/>Cell Movement<br/>Cell Proliferation<br/>Glycosylation<br/>Humans<br/>Interferon-gamma--metabolism<br/>Interleukin-10--metabolism<br/>Liver Neoplasms--enzymology<br/>Matrix Metalloproteinase 2--metabolism<br/>Matrix Metalloproteinase 7--metabolism<br/>Matrix Metalloproteinase 9--metabolism<br/>Mice<br/>N-Acetylneuraminic Acid--chemistry<br/>Sialyltransferases--metabolism<br/>Signal Transduction<br/>Transforming Growth Factor beta1--metabolism<br/>Tumor Escape<br/>Tumor Necrosis Factor-alpha--metabolism<br/>beta-D-Galactoside alpha 2-6-Sialyltransferase