Tozaki-Saitoh, Hidetoshi <br/><br/>
Transcription factor MafB contributes to the activation of spinal microglia underlying neuropathic pain development.  [electronic resource] 

 -  Glia  04 2019 
 - 729-740 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1098-1136 
<br/><br/><label>Standard No.: </label>10.1002/glia.23570  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>CD11b Antigen--metabolism<br/>Calcium-Binding Proteins--metabolism<br/>Cells, Cultured<br/>Disease Models, Animal<br/>Embryo, Mammalian<br/>Gene Expression Regulation--genetics<br/>Glial Fibrillary Acidic Protein--metabolism<br/>Green Fluorescent Proteins--genetics<br/>Hyperalgesia--genetics<br/>MafB Transcription Factor--genetics<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Transgenic<br/>Microfilament Proteins--metabolism<br/>Microglia--metabolism<br/>Neuralgia--drug therapy<br/>Pain Threshold--physiology<br/>Phosphopyruvate Hydratase--genetics<br/>RNA, Messenger--metabolism<br/>RNA, Small Interfering--therapeutic use<br/>Spinal Cord--pathology