Litvinchuk, Alexandra <br/><br/>
Complement C3aR Inactivation Attenuates Tau Pathology and Reverses an Immune Network Deregulated in Tauopathy Models and Alzheimer's Disease.  [electronic resource] 

 -  Neuron  12 2018 
 - 1337-1353.e5 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1097-4199 
<br/><br/><label>Standard No.: </label>10.1016/j.neuron.2018.10.031  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Aged<br/>Aged, 80 and over<br/>Alzheimer Disease--complications<br/>Animals<br/>Brain--pathology<br/>Calcium-Binding Proteins<br/>Cognition Disorders--etiology<br/>Complement C3a--genetics<br/>Cytokines--metabolism<br/>DNA-Binding Proteins--metabolism<br/>Disease Models, Animal<br/>Evoked Potentials--physiology<br/>Female<br/>Gene Regulatory Networks--physiology<br/>Humans<br/>Male<br/>Mice<br/>Mice, Transgenic<br/>Microfilament Proteins<br/>Middle Aged<br/>Receptors, Complement--genetics<br/>Signal Transduction--drug effects<br/>Tauopathies--complications<br/>Up-Regulation--physiology<br/>tau Proteins--genetics