Zhou, Li-Jie <br/><br/>
Erinacine Facilitates the Opening of the Mitochondrial Permeability Transition Pore Through the Inhibition of the PI3K/ Akt/GSK-3β Signaling Pathway in Human Hepatocellular Carcinoma.  [electronic resource] 

 -  Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology  2018 
 - 851-867 p.  digital 
<br/><br/> Publication Type: Journal Article; Retracted Publication 
<br/><br/><label>ISSN: </label>1421-9778 
<br/><br/><label>Standard No.: </label>10.1159/000494472  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Carcinoma, Hepatocellular--metabolism<br/>Cell Cycle Checkpoints--drug effects<br/>Chromones--pharmacology<br/>Diterpenes--pharmacology<br/>Female<br/>Glycogen Synthase Kinase 3 beta--genetics<br/>Hep G2 Cells<br/>Humans<br/>Liver Neoplasms--metabolism<br/>Male<br/>Membrane Potential, Mitochondrial--drug effects<br/>Mice, Inbred BALB C<br/>Mice, Nude<br/>Middle Aged<br/>Mitochondrial Membrane Transport Proteins--drug effects<br/>Mitochondrial Permeability Transition Pore<br/>Morpholines--pharmacology<br/>Phosphatidylinositol 3-Kinases--genetics<br/>Proto-Oncogene Proteins c-akt--genetics<br/>Signal Transduction--drug effects