Tao, Zhennan <br/><br/>
Autophagy suppresses self-renewal ability and tumorigenicity of glioma-initiating cells and promotes Notch1 degradation.  [electronic resource] 

 -  Cell death & disease  10 2018 
 - 1063 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2041-4889 
<br/><br/><label>Standard No.: </label>10.1038/s41419-018-0957-3  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antineoplastic Agents--pharmacology<br/>Autophagy--drug effects<br/>Brain Neoplasms--drug therapy<br/>Calcium-Binding Proteins--genetics<br/>Carcinogenesis--drug effects<br/>Cell Differentiation--drug effects<br/>Cell Line, Tumor<br/>Cell Proliferation--drug effects<br/>Female<br/>Gene Expression Regulation, Neoplastic<br/>Glioblastoma--drug therapy<br/>Humans<br/>Membrane Proteins--genetics<br/>Mice<br/>Mice, Nude<br/>Morpholines--pharmacology<br/>Neoplastic Stem Cells--drug effects<br/>Proteolysis--drug effects<br/>Receptor, Notch1--genetics<br/>Signal Transduction<br/>Sirolimus--pharmacology<br/>Sodium-Potassium-Exchanging ATPase--genetics<br/>Spheroids, Cellular--drug effects<br/>Survival Analysis<br/>Xenograft Model Antitumor Assays