Nanta, Rajesh <br/><br/>
Inhibition of sonic hedgehog and PI3K/Akt/mTOR pathways cooperate in suppressing survival, self-renewal and tumorigenic potential of glioblastoma-initiating cells.  [electronic resource] 

 -  Molecular and cellular biochemistry  Apr 2019 
 - 11-23 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1573-4919 
<br/><br/><label>Standard No.: </label>10.1007/s11010-018-3448-z  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antineoplastic Agents--pharmacology<br/>Biphenyl Compounds--pharmacology<br/>Cell Proliferation<br/>Glioblastoma--drug therapy<br/>Hedgehog Proteins--metabolism<br/>Humans<br/>Imidazoles--pharmacology<br/>Mice<br/>Mice, SCID<br/>Neoplastic Stem Cells--drug effects<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Phosphoinositide-3 Kinase Inhibitors<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Pyridines--pharmacology<br/>Quinolines--pharmacology<br/>Signal Transduction--drug effects<br/>Smoothened Receptor--antagonists & inhibitors<br/>TOR Serine-Threonine Kinases--antagonists & inhibitors