Translational and HIF-1α-Dependent Metabolic Reprogramming Underpin Metabolic Plasticity and Responses to Kinase Inhibitors and Biguanides. [electronic resource]
- Cell metabolism 12 2018
- 817-832.e8 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
1932-7420
10.1016/j.cmet.2018.09.001 doi
Adaptor Proteins, Signal Transducing--metabolism Amino Acids--metabolism Animals Antineoplastic Combined Chemotherapy Protocols--pharmacology Biguanides--pharmacology Cell Cycle Proteins Drug Resistance, Neoplasm Eukaryotic Initiation Factors--metabolism Gene Expression Regulation, Neoplastic HCT116 Cells Humans Hypoxia-Inducible Factor 1, alpha Subunit--metabolism K562 Cells Mechanistic Target of Rapamycin Complex 1--metabolism Mice Mice, Nude Neoplasms--drug therapy Phosphoproteins--metabolism Protein Kinase Inhibitors--pharmacology RNA, Messenger--metabolism Signal Transduction--drug effects Xenograft Model Antitumor Assays