Sugisawa, Ryoichi <br/><br/>
Independent modes of disease repair by AIM protein distinguished in AIM-felinized mice.  [electronic resource] 

 -  Scientific reports  09 2018 
 - 13157 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2045-2322 
<br/><br/><label>Standard No.: </label>10.1038/s41598-018-31580-6  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adipocytes--immunology<br/>Animals<br/>Apoptosis Regulatory Proteins--blood<br/>Carcinoma, Hepatocellular--etiology<br/>Cats<br/>Diet, High-Fat--adverse effects<br/>Disease Resistance--genetics<br/>Fatty Liver--etiology<br/>Gene Expression Regulation<br/>Hepatocytes--immunology<br/>Immunoglobulin M--blood<br/>Kupffer Cells--immunology<br/>Liver Neoplasms--etiology<br/>Mice<br/>Mice, Transgenic<br/>Obesity--etiology<br/>Protein Binding<br/>Receptors, Immunologic--blood<br/>Signal Transduction<br/>Transgenes