Wu, Hsin-Yi <br/><br/>
β-Amyloid Induces Pathology-Related Patterns of Tau Hyperphosphorylation at Synaptic Terminals.  [electronic resource] 

 -  Journal of neuropathology and experimental neurology  09 2018 
 - 814-826 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1554-6578 
<br/><br/><label>Standard No.: </label>10.1093/jnen/nly059  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alzheimer Disease--genetics<br/>Amyloid beta-Protein Precursor--genetics<br/>Animals<br/>Cerebral Cortex--pathology<br/>Chromatography, Affinity<br/>Disease Models, Animal<br/>Gene Expression Regulation--genetics<br/>Hippocampus--pathology<br/>Humans<br/>Mice<br/>Mice, Transgenic<br/>Mutation--genetics<br/>Phosphorylation<br/>Presenilin-1--genetics<br/>Synapses--metabolism<br/>Synaptosomes--metabolism<br/>tau Proteins--metabolism