Lyu, Guoliang <br/><br/>
TGF-β signaling alters H4K20me3 status via miR-29 and contributes to cellular senescence and cardiac aging.  [electronic resource] 

 -  Nature communications  07 2018 
 - 2560 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2041-1723 
<br/><br/><label>Standard No.: </label>10.1038/s41467-018-04994-z  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aging--genetics<br/>Animals<br/>Cellular Senescence--genetics<br/>DNA Methylation--genetics<br/>Embryo, Mammalian<br/>Epigenesis, Genetic<br/>Female<br/>Fibroblasts<br/>HEK293 Cells<br/>Heart--physiology<br/>Heterocyclic Compounds, 4 or More Rings--pharmacology<br/>Histone-Lysine N-Methyltransferase--antagonists & inhibitors<br/>Histones--metabolism<br/>Human Umbilical Vein Endothelial Cells<br/>Humans<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>MicroRNAs--metabolism<br/>Myocytes, Cardiac--physiology<br/>Oxidative Stress--physiology<br/>Primary Cell Culture<br/>Signal Transduction--genetics<br/>Transforming Growth Factor beta--metabolism