Saber, Sameh <br/><br/>
Perindopril, fosinopril and losartan inhibited the progression of diethylnitrosamine-induced hepatocellular carcinoma in mice via the inactivation of nuclear transcription factor kappa-B.  [electronic resource] 

 -  Toxicology letters  Oct 2018 
 - 32-40 p.  digital 
<br/><br/> Publication Type: Comparative Study; Journal Article 
<br/><br/><label>ISSN: </label>1879-3169 
<br/><br/><label>Standard No.: </label>10.1016/j.toxlet.2018.05.036  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Angiotensin II Type 1 Receptor Blockers--pharmacology<br/>Angiotensin-Converting Enzyme Inhibitors--pharmacology<br/>Animals<br/>Antineoplastic Agents--pharmacology<br/>Carcinoma, Hepatocellular--chemically induced<br/>Cyclin D1--genetics<br/>Diethylnitrosamine<br/>Fosinopril--pharmacology<br/>Liver Neoplasms, Experimental--chemically induced<br/>Losartan--pharmacology<br/>Male<br/>Matrix Metalloproteinase 2--metabolism<br/>Mice<br/>NF-KappaB Inhibitor alpha--metabolism<br/>NF-kappa B--metabolism<br/>Niacinamide--analogs & derivatives<br/>Perindopril--pharmacology<br/>Phenylurea Compounds--pharmacology<br/>Phosphorylation<br/>Renin-Angiotensin System--drug effects<br/>Signal Transduction--drug effects<br/>Sorafenib<br/>Time Factors<br/>Transcription Factor RelA--metabolism<br/>Transforming Growth Factor beta1--metabolism<br/>Tumor Necrosis Factor-alpha--metabolism<br/>Vascular Endothelial Growth Factor A--metabolism