Alawieh, Ali <br/><br/>
Targeted complement inhibition salvages stressed neurons and inhibits neuroinflammation after stroke in mice.  [electronic resource] 

 -  Science translational medicine  05 2018 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>1946-6242 
<br/><br/><label>Standard No.: </label>10.1126/scitranslmed.aao6459  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Brain Ischemia--complications<br/>Complement Activation<br/>Complement System Proteins--metabolism<br/>Disease Models, Animal<br/>Disease Susceptibility<br/>Immunoglobulin M--metabolism<br/>Infarction, Middle Cerebral Artery--complications<br/>Inflammation--complications<br/>Ischemia--pathology<br/>Mice<br/>Mice, Inbred C57BL<br/>Microglia--metabolism<br/>Nerve Degeneration--complications<br/>Neurons--drug effects<br/>Neuroprotective Agents--pharmacology<br/>Opsonin Proteins--metabolism<br/>Phagocytosis--drug effects<br/>Pneumonia--complications<br/>Proto-Oncogene Proteins c-fos--metabolism<br/>Recovery of Function--drug effects<br/>Stroke--complications<br/>Time Factors