Chen, Mo <br/><br/>
Extracellular anti-angiogenic proteins augment an endosomal protein trafficking pathway to reach mitochondria and execute apoptosis in HUVECs.  [electronic resource] 

 -  Cell death and differentiation  11 2018 
 - 1905-1920 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1476-5403 
<br/><br/><label>Standard No.: </label>10.1038/s41418-018-0092-9  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Angiostatins--genetics<br/>Apoptosis--drug effects<br/>Cell Membrane--metabolism<br/>Endocytosis<br/>Endoplasmic Reticulum Chaperone BiP<br/>Endosomes--metabolism<br/>Fibronectins--pharmacology<br/>Fluorescence Recovery After Photobleaching<br/>Heat-Shock Proteins--chemistry<br/>Human Umbilical Vein Endothelial Cells<br/>Humans<br/>Microscopy, Fluorescence<br/>Mitochondria--metabolism<br/>Neovascularization, Physiologic--drug effects<br/>Phosphoproteins--antagonists & inhibitors<br/>Protein Transport--drug effects<br/>RNA Interference<br/>RNA, Small Interfering--metabolism<br/>Recombinant Proteins--biosynthesis<br/>Sodium-Hydrogen Exchangers--antagonists & inhibitors<br/>Synaptosomal-Associated Protein 25--antagonists & inhibitors<br/>Thrombospondins--genetics