Schwab, D M <br/><br/>
AAV-9 mediated phosphatase-1 inhibitor-1 overexpression improves cardiac contractility in unchallenged mice but is deleterious in pressure-overload.  [electronic resource] 

 -  Gene therapy  01 2018 
 - 13-19 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1476-5462 
<br/><br/><label>Standard No.: </label>10.1038/gt.2017.97  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Calcium-Binding Proteins--metabolism<br/>Dependovirus<br/>Disease Models, Animal<br/>Echocardiography<br/>Gene Expression<br/>Genetic Therapy--methods<br/>Genetic Vectors<br/>Heart Failure--therapy<br/>Humans<br/>Intracellular Signaling Peptides and Proteins--genetics<br/>Mice, Inbred C57BL<br/>Mice, Transgenic<br/>Myocardial Contraction--genetics<br/>Phosphorylation<br/>Promoter Regions, Genetic<br/>Troponin T--genetics