Xu, Qingqing

Hyper-acetylation contributes to the sensitivity of chemo-resistant prostate cancer cells to histone deacetylase inhibitor Trichostatin A. [electronic resource] - Journal of cellular and molecular medicine 03 2018 - 1909-1922 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1582-4934

10.1111/jcmm.13475 doi


Acetyl Coenzyme A--metabolism
Adaptor Proteins, Signal Transducing
Allografts
Animals
Antineoplastic Agents--pharmacology
Apoptosis--drug effects
Carrier Proteins--genetics
Cell Cycle Proteins
Cell Line, Tumor
Cell Proliferation--drug effects
Cisplatin--pharmacology
Docetaxel--pharmacology
Doxorubicin--pharmacology
Drug Resistance, Neoplasm--drug effects
Endoplasmic Reticulum Stress--drug effects
Eukaryotic Initiation Factors
Gene Expression Regulation, Neoplastic
Histone Deacetylase Inhibitors--pharmacology
Histones--genetics
Humans
Hydroxamic Acids--pharmacology
Male
Mice
Mice, Inbred C57BL
Phosphoproteins--genetics
Prostatic Neoplasms--drug therapy
Protein Isoforms--genetics
Proto-Oncogene Proteins c-akt--genetics
Signal Transduction
TOR Serine-Threonine Kinases--genetics
Tumor Burden--drug effects