Zhang, Congcong <br/><br/>
Complement C3a signaling facilitates skeletal muscle regeneration by regulating monocyte function and trafficking.  [electronic resource] 

 -  Nature communications  12 2017 
 - 2078 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2041-1723 
<br/><br/><label>Standard No.: </label>10.1038/s41467-017-01526-z  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Bone Marrow Transplantation<br/>Cardiotoxins--toxicity<br/>Cell Movement--physiology<br/>Cells, Cultured<br/>Chemokine CCL5--metabolism<br/>Chimera--physiology<br/>Complement C3a--antagonists & inhibitors<br/>Complement Pathway, Alternative--physiology<br/>Disease Models, Animal<br/>Elapid Venoms--pharmacology<br/>Humans<br/>Inflammation--immunology<br/>Macrophages--physiology<br/>Male<br/>Mice<br/>Mice, Knockout<br/>Monocytes--physiology<br/>Muscle, Skeletal--drug effects<br/>Receptors, Complement--deficiency<br/>Regeneration--drug effects<br/>Signal Transduction--physiology<br/>Wound Healing--physiology