Feng, Chih-Chung <br/><br/>
Cardiac apoptosis induced under high glucose condition involves activation of IGF2R signaling in H9c2 cardiomyoblasts and streptozotocin-induced diabetic rat hearts.  [electronic resource] 

 -  Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie  Jan 2018 
 - 880-885 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1950-6007 
<br/><br/><label>Standard No.: </label>10.1016/j.biopha.2017.11.020  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--physiology<br/>Cardiomegaly--physiopathology<br/>Caspase 3--metabolism<br/>Cell Line<br/>Diabetes Mellitus, Experimental--physiopathology<br/>Down-Regulation<br/>Glucose--metabolism<br/>Hyperglycemia--complications<br/>In Situ Nick-End Labeling<br/>Insulin-Like Growth Factor II--genetics<br/>Male<br/>Myocytes, Cardiac--pathology<br/>Rats<br/>Rats, Wistar<br/>Receptor, IGF Type 2--genetics<br/>Signal Transduction<br/>Streptozocin<br/>Up-Regulation