Outer inflammatory protein a (OipA) of Helicobacter pylori is regulated by host cell contact and mediates CagA translocation and interleukin-8 response only in the presence of a functional cag pathogenicity island type IV secretion system. [electronic resource]
- Pathogens and disease 11 2017
Publication Type: Journal Article
2049-632X
10.1093/femspd/ftx113 doi
Amplified Fragment Length Polymorphism Analysis Antigens, Bacterial--genetics Bacterial Adhesion--physiology Bacterial Outer Membrane Proteins--genetics Bacterial Proteins--genetics Cell Line, Tumor Genomic Islands--genetics Helicobacter Infections--microbiology Helicobacter pylori--genetics Host-Pathogen Interactions Humans Interleukin-8--immunology Protein Transport--physiology RNA, Messenger--biosynthesis Type IV Secretion Systems--genetics