Tang, Qian <br/><br/>
Trehalose ameliorates oxidative stress-mediated mitochondrial dysfunction and ER stress via selective autophagy stimulation and autophagic flux restoration in osteoarthritis development.  [electronic resource] 

 -  Cell death & disease  10 2017 
 - e3081 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2041-4889 
<br/><br/><label>Standard No.: </label>10.1038/cddis.2017.453  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Autophagy--drug effects<br/>Chondrocytes--drug effects<br/>DNA Damage--drug effects<br/>Endoplasmic Reticulum Stress--drug effects<br/>Humans<br/>Membrane Proteins--genetics<br/>Mice<br/>Mitochondrial Proteins--genetics<br/>Osteoarthritis--drug therapy<br/>Oxidative Stress--drug effects<br/>Phosphoprotein Phosphatases--genetics<br/>RNA, Small Interfering--administration & dosage<br/>RNA-Binding Proteins--genetics<br/>Trehalose--administration & dosage<br/>tert-Butylhydroperoxide--toxicity