McLeod, Jamie Josephine Avila <br/><br/>
Didox (3,4-dihydroxybenzohydroxamic acid) suppresses IgE-mediated mast cell activation through attenuation of NFκB and AP-1 transcription.  [electronic resource] 

 -  Cellular immunology  Dec 2017 
 - 41-48 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1090-2163 
<br/><br/><label>Standard No.: </label>10.1016/j.cellimm.2017.09.008  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylcysteine--pharmacology<br/>Animals<br/>Anti-Inflammatory Agents--pharmacology<br/>Antioxidants--pharmacology<br/>Bone Marrow Cells--immunology<br/>Catalase--biosynthesis<br/>Cell Degranulation--drug effects<br/>Cells, Cultured<br/>Chemokine CCL3--biosynthesis<br/>Hydroxamic Acids--pharmacology<br/>Hypersensitivity--drug therapy<br/>Immunoglobulin E--immunology<br/>Interleukin-13--biosynthesis<br/>Interleukin-6--biosynthesis<br/>Mast Cells--drug effects<br/>Mice<br/>Mice, Inbred C57BL<br/>NF-kappa B--genetics<br/>Oxidative Stress--drug effects<br/>Reactive Oxygen Species--metabolism<br/>Superoxide Dismutase--biosynthesis<br/>Transcription Factor AP-1--genetics<br/>Transcription, Genetic--drug effects<br/>Tumor Necrosis Factor-alpha--biosynthesis