Zhang, Li <br/><br/>
Dickkopf 3 (Dkk3) Improves Amyloid-β Pathology, Cognitive Dysfunction, and Cerebral Glucose Metabolism in a Transgenic Mouse Model of Alzheimer's Disease.  [electronic resource] 

 -  Journal of Alzheimer's disease : JAD  2017 
 - 733-746 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1875-8908 
<br/><br/><label>Standard No.: </label>10.3233/JAD-161254  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adaptor Proteins, Signal Transducing<br/>Aged, 80 and over<br/>Alzheimer Disease--complications<br/>Amyloid beta-Peptides--metabolism<br/>Amyloid beta-Protein Precursor--genetics<br/>Animals<br/>Cerebral Cortex--metabolism<br/>Cognition Disorders--diagnostic imaging<br/>Disease Models, Animal<br/>Exploratory Behavior--physiology<br/>Female<br/>Gene Expression Regulation--genetics<br/>Glucose--metabolism<br/>Humans<br/>Intercellular Signaling Peptides and Proteins--genetics<br/>Male<br/>Maze Learning--physiology<br/>Mice<br/>Mice, Transgenic<br/>Mutation--genetics<br/>Presenilin-1--genetics<br/>Wnt Signaling Pathway--genetics