Giles, Daniel A <br/><br/>
Thermoneutral housing exacerbates nonalcoholic fatty liver disease in mice and allows for sex-independent disease modeling.  [electronic resource] 

 -  Nature medicine  Jul 2017 
 - 829-838 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1546-170X 
<br/><br/><label>Standard No.: </label>10.1038/nm.4346  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cold Temperature<br/>Corticosterone--metabolism<br/>Diet, High-Fat<br/>Disease Models, Animal<br/>Disease Progression<br/>Female<br/>Flow Cytometry<br/>Gastrointestinal Microbiome--immunology<br/>Gene Expression Profiling<br/>Gram-Negative Bacteria--immunology<br/>Hematopoietic Stem Cells--metabolism<br/>Housing, Animal<br/>Humans<br/>Inflammation<br/>Intestinal Mucosa--metabolism<br/>Jejunum--metabolism<br/>Machine Learning<br/>Male<br/>Mice<br/>Mice, Knockout<br/>Non-alcoholic Fatty Liver Disease--immunology<br/>Obesity--immunology<br/>Permeability<br/>Receptors, Interleukin-17--genetics<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Sex Factors<br/>Stress, Physiological--immunology<br/>Temperature<br/>Toll-Like Receptor 4--genetics