Gao, Fei <br/><br/>
Suppression of Akt1-β-catenin pathway in advanced prostate cancer promotes TGFβ1-mediated epithelial to mesenchymal transition and metastasis.  [electronic resource] 

 -  Cancer letters  08 2017 
 - 177-189 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1872-7980 
<br/><br/><label>Standard No.: </label>10.1016/j.canlet.2017.05.028  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenocarcinoma--drug therapy<br/>Animals<br/>Antineoplastic Agents--therapeutic use<br/>Cell Line, Tumor<br/>Cell Movement--drug effects<br/>Down-Regulation<br/>Epithelial-Mesenchymal Transition--drug effects<br/>Gene Expression Regulation, Neoplastic<br/>Genetic Predisposition to Disease<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mice, Nude<br/>Phenotype<br/>Prostatic Neoplasms--drug therapy<br/>Protein Kinase Inhibitors--therapeutic use<br/>Protein Serine-Threonine Kinases--metabolism<br/>Proto-Oncogene Proteins c-akt--antagonists & inhibitors<br/>RNA Interference<br/>Receptor, Transforming Growth Factor-beta Type II<br/>Receptors, Transforming Growth Factor beta--metabolism<br/>Signal Transduction<br/>Time Factors<br/>Transfection<br/>Transforming Growth Factor beta1--metabolism<br/>Xenograft Model Antitumor Assays<br/>beta Catenin--antagonists & inhibitors