Hahn, M <br/><br/>
Aberrant splicing of the tumor suppressor CYLD promotes the development of chronic lymphocytic leukemia via sustained NF-κB signaling.  [electronic resource] 

 -  Leukemia  01 2018 
 - 72-82 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1476-5551 
<br/><br/><label>Standard No.: </label>10.1038/leu.2017.168  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>B-Lymphocytes--metabolism<br/>CD5 Antigens--genetics<br/>Cell Proliferation--genetics<br/>Cell Survival--genetics<br/>Deubiquitinating Enzyme CYLD--genetics<br/>Genes, Tumor Suppressor--physiology<br/>Humans<br/>Leukemia, Lymphocytic, Chronic, B-Cell--genetics<br/>Mice<br/>NF-kappa B--genetics<br/>RNA Splicing--genetics<br/>Signal Transduction--genetics<br/>Ubiquitination--genetics