Lin, Mei-Yao <br/><br/>
Releasing Syntaphilin Removes Stressed Mitochondria from Axons Independent of Mitophagy under Pathophysiological Conditions.  [electronic resource] 

 -  Neuron  May 2017 
 - 595-610.e6 p.  digital 
<br/><br/> Publication Type: Journal Article; Video-Audio Media 
<br/><br/><label>ISSN: </label>1097-4199 
<br/><br/><label>Standard No.: </label>10.1016/j.neuron.2017.04.004  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aged<br/>Aged, 80 and over<br/>Alzheimer Disease--genetics<br/>Amyotrophic Lateral Sclerosis--genetics<br/>Animals<br/>Axons--metabolism<br/>Calcium--metabolism<br/>Carrier Proteins--metabolism<br/>Cerebral Cortex--cytology<br/>Endosomes--metabolism<br/>Female<br/>Humans<br/>Male<br/>Membrane Proteins<br/>Mice<br/>Mice, Knockout<br/>Microscopy, Immunoelectron<br/>Microtubule-Associated Proteins--metabolism<br/>Middle Aged<br/>Mitochondria--metabolism<br/>Mitophagy--genetics<br/>Motor Neurons--metabolism<br/>Mutation<br/>Nerve Tissue Proteins--metabolism<br/>Neurons--cytology<br/>Rats<br/>Spinal Cord--cytology<br/>Stress, Physiological<br/>Ubiquitin-Protein Ligases--genetics<br/>Vesicular Transport Proteins--metabolism