Bongiorno, Emily K <br/><br/>
Type 1 Immune Mechanisms Driven by the Response to Infection with Attenuated Rabies Virus Result in Changes in the Immune Bias of the Tumor Microenvironment and Necrosis of Mouse GL261 Brain Tumors.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  06 2017 
 - 4513-4523 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1550-6606 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.1601444  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>B-Lymphocytes--immunology<br/>Blood-Brain Barrier--immunology<br/>Brain--immunology<br/>Brain Neoplasms--immunology<br/>CD4-Positive T-Lymphocytes<br/>Cytokines--immunology<br/>Disease Models, Animal<br/>Female<br/>Interferon-gamma--biosynthesis<br/>Mice<br/>Necrosis--virology<br/>Nitric Oxide Synthase Type II--biosynthesis<br/>Rabies virus--genetics<br/>T-Box Domain Proteins--deficiency<br/>Th2 Cells--immunology<br/>Tumor Microenvironment--immunology<br/>Tumor Necrosis Factor-alpha--biosynthesis<br/>T-bet Transcription Factor