Kim, Hwisung <br/><br/>
Polyamidoamine dendrimer-conjugated triamcinolone acetonide attenuates nerve injury-induced spinal cord microglia activation and mechanical allodynia.  [electronic resource] 

 -  Molecular pain  01 2017 
 - 1744806917697006 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1744-8069 
<br/><br/><label>Standard No.: </label>10.1177/1744806917697006  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Anti-Inflammatory Agents--pharmacology<br/>Calcium-Binding Proteins--metabolism<br/>Cells, Cultured<br/>Cytokines--metabolism<br/>Dendrimers--chemistry<br/>Disease Models, Animal<br/>Dose-Response Relationship, Drug<br/>Glial Fibrillary Acidic Protein--metabolism<br/>Hyperalgesia--etiology<br/>Male<br/>Membrane Glycoproteins--metabolism<br/>Mice<br/>Mice, Inbred C57BL<br/>Microfilament Proteins--metabolism<br/>Microglia--drug effects<br/>NADPH Oxidase 2<br/>NADPH Oxidases--metabolism<br/>Peripheral Nerve Injuries--complications<br/>Spinal Cord--pathology<br/>Triamcinolone Acetonide--chemistry