Hu, Shi <br/><br/>
Antagonism of EGFR and Notch limits resistance to EGFR inhibitors and radiation by decreasing tumor-initiating cell frequency.  [electronic resource] 

 -  Science translational medicine  03 2017 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1946-6242 
<br/><br/><label>Standard No.: </label>10.1126/scitranslmed.aag0339  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aldehyde Dehydrogenase--metabolism<br/>Animals<br/>Antibodies--pharmacology<br/>Antineoplastic Agents--pharmacology<br/>Carcinogenesis--pathology<br/>Carcinoma, Non-Small-Cell Lung--drug therapy<br/>Cell Count<br/>Cell Line, Tumor<br/>Cell Proliferation--drug effects<br/>Drug Resistance, Neoplasm--drug effects<br/>Epithelial-Mesenchymal Transition--drug effects<br/>ErbB Receptors--antagonists & inhibitors<br/>Lung Neoplasms--drug therapy<br/>Mice, SCID<br/>Molecular Targeted Therapy<br/>Neoplasm Recurrence, Local--drug therapy<br/>Neoplastic Stem Cells--drug effects<br/>Protein Kinase Inhibitors--pharmacology<br/>Radiation Tolerance--drug effects<br/>Receptors, Notch--antagonists & inhibitors<br/>Signal Transduction--drug effects