Wang, Yandao <br/><br/>
β-Arrestin-biased AT1R stimulation promotes extracellular matrix synthesis in renal fibrosis.  [electronic resource] 

 -  American journal of physiology. Renal physiology  07 2017 
 - F1-F8 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1522-1466 
<br/><br/><label>Standard No.: </label>10.1152/ajprenal.00588.2016  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Angiotensin II--analogs & derivatives<br/>Angiotensin II Type 1 Receptor Blockers--pharmacology<br/>Animals<br/>Benzimidazoles--pharmacology<br/>Biphenyl Compounds<br/>Cell Line<br/>Collagen Type I--metabolism<br/>Disease Models, Animal<br/>Extracellular Matrix--metabolism<br/>Fibroblasts--drug effects<br/>Fibronectins--metabolism<br/>Fibrosis<br/>Kidney--drug effects<br/>Kidney Diseases--etiology<br/>Male<br/>Mice, Inbred C57BL<br/>Mitogen-Activated Protein Kinase 1--metabolism<br/>Mitogen-Activated Protein Kinase 3--metabolism<br/>Phosphorylation<br/>RNA Interference<br/>Rats<br/>Receptor, Angiotensin, Type 1--drug effects<br/>Signal Transduction<br/>Tetrazoles--pharmacology<br/>Time Factors<br/>Transfection<br/>Ureteral Obstruction--complications<br/>beta-Arrestin 1--agonists<br/>beta-Arrestin 2--agonists