Chen, Zuolei <br/><br/>
Morphine Postconditioning Protects Against Reperfusion Injury: the Role of Protein Kinase C-Epsilon, Extracellular Signal-Regulated Kinase 1/2 and Mitochondrial Permeability Transition Pores.  [electronic resource] 

 -  Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology  2016 
 - 1930-1940 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1421-9778 
<br/><br/><label>Standard No.: </label>10.1159/000447890  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Creatine Kinase, MB Form--metabolism<br/>Cytochromes c--metabolism<br/>Gene Expression Regulation<br/>Ischemic Postconditioning--methods<br/>Male<br/>Mitochondrial Membrane Transport Proteins--genetics<br/>Mitochondrial Permeability Transition Pore<br/>Mitogen-Activated Protein Kinase 1--genetics<br/>Mitogen-Activated Protein Kinase 3--genetics<br/>Morphine--antagonists & inhibitors<br/>Myocardial Reperfusion Injury--genetics<br/>Organ Culture Techniques<br/>Peptide Fragments--pharmacology<br/>Phosphorylation--drug effects<br/>Protein Kinase C-epsilon--genetics<br/>Protein Transport--drug effects<br/>Rats<br/>Rats, Sprague-Dawley<br/>Signal Transduction