The m-AAA Protease Associated with Neurodegeneration Limits MCU Activity in Mitochondria. [electronic resource]
- Molecular cell 10 2016
- 148-162 p. digital
Publication Type: Journal Article
1097-4164
10.1016/j.molcel.2016.08.020 doi
ATP-Dependent Proteases--genetics ATPases Associated with Diverse Cellular Activities Animals Calcium--metabolism Calcium Channels--genetics Calcium-Calmodulin-Dependent Protein Kinase Type 2--genetics Cell Death Cerebellum--metabolism Corpus Striatum--metabolism Gene Expression Regulation HEK293 Cells Hippocampus--metabolism Homeostasis--genetics Humans Ion Transport Metalloendopeptidases--deficiency Mice Mice, Inbred C57BL Mice, Transgenic Mitochondria--metabolism Mitochondrial Membrane Transport Proteins--genetics Mitochondrial Permeability Transition Pore Neurons--metabolism Protein Interaction Mapping Signal Transduction