Heyne, Galen W <br/><br/>
Gli2 gene-environment interactions contribute to the etiological complexity of holoprosencephaly: evidence from a mouse model.  [electronic resource] 

 -  Disease models & mechanisms  11 2016 
 - 1307-1315 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1754-8411 
<br/><br/><label>Standard No.: </label>10.1242/dmm.026328  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Body Patterning<br/>Brain--abnormalities<br/>Disease Models, Animal<br/>Face--abnormalities<br/>Fetus--abnormalities<br/>Gene-Environment Interaction<br/>Hedgehog Proteins--metabolism<br/>Heterozygote<br/>Holoprosencephaly--embryology<br/>Ligands<br/>Loss of Function Mutation--genetics<br/>Male<br/>Mice, Inbred C57BL<br/>Teratogens--toxicity<br/>Zinc Finger Protein Gli2--genetics