Liu, Chun-Wei <br/><br/>
Rosuvastatin postconditioning protects isolated hearts against ischemia-reperfusion injury: The role of radical oxygen species, PI3K-Akt-GSK-3β pathway, and mitochondrial permeability transition pore.  [electronic resource] 

 -  Cardiovascular therapeutics  Feb 2017 
 - 3-9 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1755-5922 
<br/><br/><label>Standard No.: </label>10.1111/1755-5922.12225  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antioxidants--pharmacology<br/>Calcium Signaling--drug effects<br/>Cytoprotection<br/>Disease Models, Animal<br/>Dose-Response Relationship, Drug<br/>Glycogen Synthase Kinase 3 beta--metabolism<br/>Isolated Heart Preparation<br/>Male<br/>Mitochondria, Heart--drug effects<br/>Mitochondrial Membrane Transport Proteins--antagonists & inhibitors<br/>Mitochondrial Permeability Transition Pore<br/>Myocardial Infarction--enzymology<br/>Myocardial Reperfusion Injury--enzymology<br/>Myocytes, Cardiac--drug effects<br/>Oxidative Stress--drug effects<br/>Phosphatidylinositol 3-Kinase--metabolism<br/>Phosphoinositide-3 Kinase Inhibitors<br/>Phosphorylation<br/>Protein Kinase Inhibitors--pharmacology<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Rats, Wistar<br/>Reactive Oxygen Species--metabolism<br/>Rosuvastatin Calcium--pharmacology<br/>Time Factors<br/>Ventricular Function, Left--drug effects