Lee, Wei-Ping <br/><br/>
Helicobacter pylori-induced chronic inflammation causes telomere shortening of gastric mucosa by promoting PARP-1-mediated non-homologous end joining of DNA.  [electronic resource] 

 -  Archives of biochemistry and biophysics  09 2016 
 - 90-8 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1096-0384 
<br/><br/><label>Standard No.: </label>10.1016/j.abb.2016.07.014  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Chronic Disease<br/>DNA--chemistry<br/>DNA Damage<br/>DNA End-Joining Repair<br/>Female<br/>Gastric Mucosa--metabolism<br/>Gastritis--metabolism<br/>Helicobacter Infections--drug therapy<br/>Helicobacter pylori--pathogenicity<br/>Histones--chemistry<br/>Humans<br/>Hydrogen Peroxide--chemistry<br/>Inflammation--microbiology<br/>Male<br/>Middle Aged<br/>NF-kappa B--metabolism<br/>Oxidative Stress<br/>Oxygen--chemistry<br/>Poly (ADP-Ribose) Polymerase-1--metabolism<br/>RNA, Small Interfering--metabolism<br/>Telomere--chemistry