Hackstein, Carl-Philipp <br/><br/>
Gut microbial translocation corrupts myeloid cell function to control bacterial infection during liver cirrhosis.  [electronic resource] 

 -  Gut  03 2017 
 - 507-518 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1468-3288 
<br/><br/><label>Standard No.: </label>10.1136/gutjnl-2015-311224  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Bacterial Translocation<br/>Carbon Tetrachloride<br/>Immunity, Innate--genetics<br/>Interferon Type I--metabolism<br/>Interleukin-10--biosynthesis<br/>Listeriosis--complications<br/>Liver Cirrhosis, Experimental--complications<br/>Membrane Glycoproteins--genetics<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mice, Transgenic<br/>Myeloid Cells--immunology<br/>Myxovirus Resistance Proteins--genetics<br/>Receptor, Interferon alpha-beta--antagonists & inhibitors<br/>Receptors, Interleukin-10--antagonists & inhibitors<br/>Receptors, Pattern Recognition--genetics<br/>Signal Transduction<br/>Toll-Like Receptor 2--genetics<br/>Toll-Like Receptor 4--genetics<br/>Toll-Like Receptor 7--genetics<br/>Toll-Like Receptor 9--genetics