Shimizu, Toshiaki <br/><br/>
HMGB1 is secreted by 3T3-L1 adipocytes through JNK signaling and the secretion is partially inhibited by adiponectin.  [electronic resource] 

 -  Obesity (Silver Spring, Md.)  09 2016 
 - 1913-21 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1930-739X 
<br/><br/><label>Standard No.: </label>10.1002/oby.21549  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>3T3-L1 Cells--metabolism<br/>Adipocytes--metabolism<br/>Adipokines--metabolism<br/>Adiponectin--metabolism<br/>Adipose Tissue--metabolism<br/>Animals<br/>Cell Differentiation--drug effects<br/>HMGB1 Protein--metabolism<br/>Humans<br/>Inflammation--metabolism<br/>Insulin--metabolism<br/>Insulin Resistance--physiology<br/>MAP Kinase Signaling System--physiology<br/>Mice<br/>Obesity--metabolism<br/>Tumor Necrosis Factor-alpha--metabolism